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The antibody against ME2 was raised in Rabbit using the recombinant fusion protein containing a sequence corresponding to amino acids 220-479 of human ME2 (NP_001161807.1) as the immunogen. The polyclonal antibody exists as a isotype IgG, by affinity purification. This antibody has been validated on WB, ELISA.
The antibody against ME2 was raised in Rabbit using the recombinant fusion protein containing a sequence corresponding to amino acids 220-479 of human ME2 (NP_001161807.1) as the immunogen. The polyclonal antibody exists as a isotype IgG, by affinity purification. This antibody has been validated on WB, ELISA.
| Cat.No | ADA-00953A | Clonality | Polyclonal |
|---|---|---|---|
| Host Species | Rabbit | Target Name | ME2 |
| Target Synonyms | ODS1; ME2 | Form | Liquid |
| Species Reactivity | Human | Isotype | IgG |
| Storage Buffer | 50% Glycerol, PBS with 0.02% sodium azide, pH7.3. | Purification Method | Affinity purification |
| Positive Samples | HL-60, HT-1080, LO2, SKOV3 | Application | ELISA, WB |
| Immunogen Description | Recombinant fusion protein containing a sequence corresponding to amino acids 220-479 of human ME2 (NP_001161807.1). | Target Species | Human |
|---|---|---|---|
| Uniprot ID | P23368 | Immunogen Sequence |
Uniprot Id
P23368
Target Species
Human
Target Name
ME2
Target Full Name
NAD-dependent malic enzyme, mitochondrial
Target Subcellular Location
Mitochondrion matrix.
Target Protein Families
Malic enzymes family
Target Synonyms
Malate dehydrogenase; Malic enzyme 2; Malic enzyme 2 mitochondrial; Malic enzyme 2 NAD(+) dependent mitochondrial; Malic enzyme mitochondrial; Malic enzyme NAD(+) dependent mitochondrial; MAOM_HUMAN; ME 2; ME2; mitochondrial; NAD dependent malic enzyme mitochondrial; NAD ME; NAD-dependent malic enzyme; NAD-ME; ODS1; Pyruvic malic carboxylase
Target Background
This gene encodes a mitochondrial NAD-dependent malic enzyme, a homotetrameric protein, that catalyzes the oxidative decarboxylation of malate to pyruvate. It had previously been weakly linked to a syndrome known as Friedreich ataxia that has since been shown to be the result of mutation in a completely different gene. Certain single-nucleotide polymorphism haplotypes of this gene have been shown to increase the risk for idiopathic generalized epilepsy. Alternatively spliced transcript variants encoding different isoforms found for this gene.
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