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The antibody against GTPBP1 was raised in Rabbit using the recombinant fusion protein containing a sequence corresponding to amino acids 10-280 of human GTPBP1 (NP_004277.2) as the immunogen. The polyclonal antibody exists as a isotype IgG, by affinity purification. This antibody has been validated on WB, ELISA.
The antibody against GTPBP1 was raised in Rabbit using the recombinant fusion protein containing a sequence corresponding to amino acids 10-280 of human GTPBP1 (NP_004277.2) as the immunogen. The polyclonal antibody exists as a isotype IgG, by affinity purification. This antibody has been validated on WB, ELISA.
| Cat.No | ADA-05112A | Clonality | Polyclonal |
|---|---|---|---|
| Host Species | Rabbit | Target Name | GTPBP1 |
| Target Synonyms | GP1; GP-1; HSPC018; GTPBP1 | Form | Liquid |
| Species Reactivity | Human, Mouse | Isotype | IgG |
| Storage Buffer | 50% Glycerol, PBS with 0.01% thimerosal, pH7.3. | Purification Method | Affinity purification |
| Positive Samples | HeLa, Jurkat, LO2, Mouse thymus, U-251MG | Application | ELISA, WB |
| Immunogen Description | Recombinant fusion protein containing a sequence corresponding to amino acids 10-280 of human GTPBP1 (NP_004277.2). | Target Species | Human |
|---|---|---|---|
| Uniprot ID | O00178 | Immunogen Sequence |
Uniprot Id
O00178
Target Species
Human
Target Name
GTPBP1
Target Full Name
GTP-binding protein 1
Target Function
Promotes degradation of target mRNA species. Plays a role in the regulation of circadian mRNA stability. Binds GTP and has GTPase activity.
Target Subcellular Location
Cytoplasm.
Target Protein Families
TRAFAC class translation factor GTPase superfamily, Classic translation factor GTPase family, GTPBP1 subfamily
Target Synonyms
G protein 1; G-protein 1; GP 1; GP-1; GP1; GTP binding protein 1; GTP-binding protein 1; GTPB1_HUMAN; GTPBP1
Target Background
This gene is upregulated by interferon-gamma and encodes a protein that is a member of the AGP11/GTPBP1 family of GTP-binding proteins. A structurally similar protein has been found in mouse, where disruption of the gene for that protein had no observable phenotype.
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