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Rabbit anti-Human SOX14 Polyclonal Antibody

The antibody against SOX14 was raised in rabbit using the Recombinant Human Transcription factor SOX-14 protein (1-137AA) as the immunogen. This antibody exists as a non-conjugated isotype IgG, purified by protein G with a purity greater than 95%. This antibody has been validated on ELISA, WB, IHC, IF.

ADC-15693A

The antibody against SOX14 was raised in rabbit using the Recombinant Human Transcription factor SOX-14 protein (1-137AA) as the immunogen. This antibody exists as a non-conjugated isotype IgG, purified by protein G with a purity greater than 95%. This antibody has been validated on ELISA, WB, IHC, IF.

$299.00

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High Purity LevelsPrecision and ReliabilityCustomization Options

Specifications


Cat.No ADC-15693A ClonalityPolyclonal
Host SpeciesRabbitTarget NameSOX14
Target SynonymsSOX14 antibody; SOX28 antibody; Transcription factor SOX-14 antibody; Protein SOX-28 antibodyFormLiquid
Species ReactivityHumanIsotypeIgG
Storage Buffer0.01M PBS, 0.03% Proclin 300; Constituents: 50% Glycerol, PH 7.4Purification Method>95%, Protein G purified
ConjugateNon-conjugatedApplicationELISA, IF, IHC, WB
StorageUpon receipt

Immunogen Information


Immunogen DescriptionRecombinant Human Transcription factor SOX-14 protein (1-137AA)Target SpeciesHuman
Immunogen SequenceComplete sequences for the immunogen, target protein, and peptides are available upon request.Uniprot IDO95416
Background Information
  • Uniprot Id

    O95416

  • Target Species

    Human

  • Target Name

    SOX14

  • Target Full Name

    Transcription factor SOX-14

  • Target Function

    Acts as a negative regulator of transcription.

  • Target Subcellular Location

    Nucleus.

  • Target Synonyms

    SOX14; SOX28; Transcription factor SOX-14; Protein SOX-28

  • Target Background

    This intronless gene encodes a member of the SOX (SRY-related HMG-box) family of transcription factors involved in the regulation of embryonic development and in the determination of the cell fate. The encoded protein may act as a transcriptional regulator after forming a protein complex with other proteins. Mutations in this gene are suggested to be responsible for the limb defects associated with blepharophimosis, ptosis, epicanthus inversus syndrome (BPES) and Mobius syndrome.

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