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Rabbit anti-Human MYO1A Polyclonal Antibody

The antibody against MYO1A was raised in rabbit using the Human MYO1A as the immunogen. This antibody exists as a non-conjugated isotype IgG, Antigen affinity purified. This antibody has been validated on ELISA, WB, IHC, IF.

ADC-52644A

The antibody against MYO1A was raised in rabbit using the Human MYO1A as the immunogen. This antibody exists as a non-conjugated isotype IgG, Antigen affinity purified. This antibody has been validated on ELISA, WB, IHC, IF.

$600.00

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Specifications


Cat.No ADC-52644A ClonalityPolyclonal
Host SpeciesRabbitTarget NameMYO1A
Target Synonymsheavy polypeptide like (100kD) antibody; Myosin-IA antibodyFormLiquid
Species ReactivityHuman, Mouse, RatIsotypeIgG
Storage Buffer50% Glycerol, Avoid freeze / thaw cycles., PBS with 0.1% Sodium AzidePurification MethodAntigen affinity purified
ConjugateNon-conjugatedApplicationELISA, IF, IHC, WB
StorageUpon receipt

Immunogen Information


Immunogen DescriptionHuman MYO1ATarget SpeciesHuman
Immunogen SequenceComplete sequences for the immunogen, target protein, and peptides are available upon request.Uniprot IDQ9UBC5
Background Information
  • Uniprot Id

    Q9UBC5

  • Target Species

    Human

  • Target Name

    MYO1A

  • Target Full Name

    Unconventional myosin-Ia

  • Target Function

    Involved in directing the movement of organelles along actin filaments.

  • Target Protein Families

    TRAFAC class myosin-kinesin ATPase superfamily, Myosin family

  • Target Synonyms

    BBM I; BBM-I; BBMI; Brush border myosin I; DFNA48; MIHC; MYHL; Myo1a; MYO1A_HUMAN; Myosin I heavy chain; Myosin, heavy polypeptide like (100kD); Myosin-IA

  • Target Background

    This gene encodes a member of the myosin superfamily. The protein represents an unconventional myosin; it should not be confused with the conventional skeletal muscle myosin-1 (MYH1). Unconventional myosins contain the basic domains characteristic of conventional myosins and are further distinguished from class members by their tail domains. They function as actin-based molecular motors. Mutations in this gene have been associated with autosomal dominant deafness. Alternatively spliced variants have been found for this gene.

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