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The antibody against CLN6 was raised in rabbit using the Recombinant Human Ceroid-lipofuscinosis neuronal protein 6 protein (1-43AA) as the immunogen. This antibody exists as a non-conjugated isotype IgG, purified by protein G with a purity greater than 95%. This antibody has been validated on ELISA, IHC.
The antibody against CLN6 was raised in rabbit using the Recombinant Human Ceroid-lipofuscinosis neuronal protein 6 protein (1-43AA) as the immunogen. This antibody exists as a non-conjugated isotype IgG, purified by protein G with a purity greater than 95%. This antibody has been validated on ELISA, IHC.
$299.00
Immunohistochemistry of paraffin-embedded human kidney tissue using CLN6 Antibody at dilution of 1:100
| Cat.No | ADC-08118A | Clonality | Polyclonal |
|---|---|---|---|
| Host Species | Rabbit | Target Name | CLN6 |
| Target Synonyms | CLN6; Ceroid-lipofuscinosis neuronal protein 6; Protein CLN6 | Form | Liquid |
| Species Reactivity | Human | Isotype | IgG |
| Storage Buffer | 0.01M PBS, 0.03% Proclin 300; Constituents: 50% Glycerol, PH 7.4 | Purification Method | >95%, Protein G purified |
| Conjugate | Non-conjugated | Application | ELISA, IHC |
| Storage | Upon receipt |
| Immunogen Description | Recombinant Human Ceroid-lipofuscinosis neuronal protein 6 protein (1-43AA) | Target Species | Human |
|---|---|---|---|
| Immunogen Sequence | Complete sequences for the immunogen, target protein, and peptides are available upon request. | Uniprot ID | Q9NWW5 |
Uniprot Id
Q9NWW5
Target Species
Human
Target Name
CLN6
Target Full Name
Ceroid-lipofuscinosis neuronal protein 6
Target Involvement
Ceroid lipofuscinosis, neuronal, 6 (CLN6); Ceroid lipofuscinosis, neuronal, 4A (CLN4A)
Target Subcellular Location
Endoplasmic reticulum membrane; Multi-pass membrane protein. Endoplasmic reticulum.
Target Synonyms
CLN6; Ceroid-lipofuscinosis neuronal protein 6; Protein CLN6
Target Background
This gene is one of eight which have been associated with neuronal ceroid lipofuscinoses (NCL). Also referred to as Batten disease, NCL comprises a class of autosomal recessive, neurodegenerative disorders affecting children. The genes responsible likely encode proteins involved in the degradation of post-translationally modified proteins in lysosomes. The primary defect in NCL disorders is thought to be associated with lysosomal storage function.
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