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The antibody against GTPBP1 was raised in rabbit using the Human GTPBP1 as the immunogen. This antibody exists as a non-conjugated isotype IgG, Antigen affinity purified. This antibody has been validated on ELISA, WB.
The antibody against GTPBP1 was raised in rabbit using the Human GTPBP1 as the immunogen. This antibody exists as a non-conjugated isotype IgG, Antigen affinity purified. This antibody has been validated on ELISA, WB.
$600.00
| Cat.No | ADC-45963A | Clonality | Polyclonal |
|---|---|---|---|
| Host Species | Rabbit | Target Name | GTPBP1 |
| Target Synonyms | G protein 1 antibody; G-protein 1 antibody; GP 1 antibody; GP-1 antibody; GP1 antibody; GTP binding protein 1 antibody; GTP-binding protein 1 antibody; GTPB1_HUMAN antibody; GTPBP1 antibody | Form | Liquid |
| Species Reactivity | Human, Mouse, Rat | Isotype | IgG |
| Storage Buffer | 50% Glycerol, Avoid freeze / thaw cycles., PBS with 0.02% sodium azide | Purification Method | Antigen affinity purified |
| Conjugate | Non-conjugated | Application | ELISA, WB |
| Storage | Upon receipt |
| Immunogen Description | Human GTPBP1 | Target Species | Human |
|---|---|---|---|
| Immunogen Sequence | Complete sequences for the immunogen, target protein, and peptides are available upon request. | Uniprot ID | O00178 |
Uniprot Id
O00178
Target Species
Human
Target Name
GTPBP1
Target Full Name
GTP-binding protein 1
Target Function
Promotes degradation of target mRNA species. Plays a role in the regulation of circadian mRNA stability. Binds GTP and has GTPase activity.
Target Subcellular Location
Cytoplasm.
Target Protein Families
TRAFAC class translation factor GTPase superfamily, Classic translation factor GTPase family, GTPBP1 subfamily
Target Synonyms
G protein 1; G-protein 1; GP 1; GP-1; GP1; GTP binding protein 1; GTP-binding protein 1; GTPB1_HUMAN; GTPBP1
Target Background
This gene is upregulated by interferon-gamma and encodes a protein that is a member of the AGP11/GTPBP1 family of GTP-binding proteins. A structurally similar protein has been found in mouse, where disruption of the gene for that protein had no observable phenotype.
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