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The antibody against TTC7A was raised in rabbit using the Human TTC7A as the immunogen. This antibody exists as a non-conjugated isotype IgG, Antigen affinity purified. This antibody has been validated on ELISA, WB.
The antibody against TTC7A was raised in rabbit using the Human TTC7A as the immunogen. This antibody exists as a non-conjugated isotype IgG, Antigen affinity purified. This antibody has been validated on ELISA, WB.
$600.00
| Cat.No | ADC-46826A | Clonality | Polyclonal |
|---|---|---|---|
| Host Species | Rabbit | Target Name | TTC7A |
| Target Synonyms | TTC7A antibody; KIAA1140 antibody; TTC7Tetratricopeptide repeat protein 7A antibody; TPR repeat protein 7A antibody | Form | Liquid |
| Species Reactivity | Human, Mouse, Rat | Isotype | IgG |
| Storage Buffer | 50% Glycerol, Avoid freeze / thaw cycles., PBS with 0.02% sodium azide | Purification Method | Antigen affinity purified |
| Conjugate | Non-conjugated | Application | ELISA, WB |
| Storage | Upon receipt |
| Immunogen Description | Human TTC7A | Target Species | Human |
|---|---|---|---|
| Immunogen Sequence | Complete sequences for the immunogen, target protein, and peptides are available upon request. | Uniprot ID | Q9ULT0 |
Uniprot Id
Q9ULT0
Target Species
Human
Target Name
TTC7A
Target Full Name
Tetratricopeptide repeat protein 7A
Target Function
Component of a complex required to localize phosphatidylinositol 4-kinase (PI4K) to the plasma membrane. The complex acts as a regulator of phosphatidylinositol 4-phosphate (PtdIns(4)P) synthesis. In the complex, plays a central role in bridging PI4KA to EFR3B and FAM126A, via direct interactions.
Target Involvement
Gastrointestinal defects and immunodeficiency syndrome (GIDID)
Target Subcellular Location
Cytoplasm. Cell membrane.
Target Tissue Specificity
Expressed in epithelial cells of the intestine, thymus, and pancreas (at protein level).
Target Synonyms
TTC7A; KIAA1140; TTC7Tetratricopeptide repeat protein 7A; TPR repeat protein 7A
Target Background
This gene encodes a protein containing tetratricopeptide repeats. Mutations in this gene disrupt intestinal development and can cause early onset inflammatory bowel disease and intestinal atresia. Alternative splicing results in multiple transcript variants.
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